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As indicated above (and in Table 28-4), the wide variety and seemingly random movements displayed by the healthy neonate are from birth, and certainly within days, rmly organized into re exive-instinctual patterns called automatisms. The most testable of the automatisms are blinking in response to light, tonic deviation of the eyes in response to labyrinthine stimulation (turning of the head), prehensile and sucking movements of the lips in response to labial contact, swallowing, avoidance movements of the head and neck, startle reaction (Moro response) in response to loud noise or dropping of the head into an extended position, grasp re exes, and support, stepping, and placing movements. This repertoire of movements, as mentioned earlier, depends on re exes organized at the spinal and brainstem levels. Only the placing reactions, ocular xation, and following movements (the latter are established by the third month) are thought to depend on emerging cortical connec-

Figure 11-3. (1) Costovertebral angle. (2) Spinous process and interspinous ligament. (3) Region of articular facet ( fth lumbar to rst sacral). (4) Dorsum of sacrum. (5) Region of iliac crest. (6) Iliolumbar angle. (7) Spinous processes of fth lumbar and rst sacral vertebrae (tenderness faulty posture or occasionally spina bi da occulta). (8) Region between posterior superior and posteroinferior spines. Sacroiliac ligaments (tenderness sacroiliac sprain, often tender, with fth lumbar or rst sacral disc). (9) Sacrococcygeal junction (tenderness sacrococcygeal injury; i.e., sprain or fracture). (10) Region of sacrosciatic notch (tenderness fourth or fth lumbar disc rupture and sacroiliac sprain). (11) Sciatic nerve trunk (tenderness ruptured lumbar disc or sciatic nerve lesion).

6:

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mediotemporal infarction, re ecting a proximal occlusion of the posterior cerebral artery Simultanagnosia Wolpert described a patient who demonstrated a spelling dyslexia (an inability to read all but the shortest words, spelled out letter by letter) and a failure to perceive simultaneously all the elements of a scene and to properly interpret the scene In the framework of gestalt psychology, the patient could recognize the parts but not the whole A cognitive defect of synthesis of the visual impressions was thought to be the basis of this condition, which Wolpert called simultanagnosia (a term that appears often in discussion) Some patients with this disorder have a right homonymous hemianopia; in others, the visual elds are full but there is one-sided extinction when tested with double simultaneous stimulation.

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This is part of the Balint syndrome (see below), the other components of which are faulty visual scanning (ocular apraxia) and visual reaching (optic ataxia), suggesting that perhaps a fault in ocular scanning might underlie all the defects Through tachistoscopic testing, Kinsbourne and Warrington found that reducing the time of stimulus exposure permits single objects to be perceived, but not two objects Rizzo and Robin have proposed that the primary defect is in sustained attention to incoming visuospatial information Nielsen has attributed this disorder to a lesion of the inferolateral part of the dominant occipital lobe (area 18) In a patient who presented with an isolated spelling dyslexia and simultanagnosia, Kinsbourne and Warrington found the lesion to be localized within the inferior part of the left occipital lobe In other instances the lesions have been bilateral in the superior parts of the occipital association cortices Balint Syndrome (See also Chap.

13) In this not uncommon syndrome, the illusion of a coherent and detailed visual world is disrupted and the patient perceives only disconnected individual parts of the scene It consists of (1) a disorder of visual attention mainly to the periphery of the visual eld, in which the totality of a scene is not percevied despite preservation of vision for individual elements, attention to other sensory stimuli being well preserved; (2) dif culty in grasping or touching an object under visual guidance, as though hand and eye were not coordinated (inappropriately called optic ataxia ); and (3) an inability to project gaze voluntarily into the peripheral eld and to scan it despite the fact that eye movements are full (psychic paralysis of xation of gaze, incorrectly called optic apraxia, since visual scanning is not a learned task) Balint, a Hungarian neurologist, was the rst to recognize this constellation.

Part II:

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